T lymphocytes are direct, aryl hydrocarbon receptor (AhR)-dependent targets of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD): AhR expression in both CD4⁺ and CD8⁺ T cells is necessary for full suppression of a cytotoxic T lymphocyte response by TCDD

Nancy I. Kerkvliet, David M. Shepherd, Linda Baecher-Steppan

Biomedical and Pharmaceutical Sciences
Center for Translational Medicine

Oregon State University

Research output: Contribution to journal › Review article › peer-review

105 Scopus citations

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T lymphocytes are direct, aryl hydrocarbon receptor (AhR)-dependent targets of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD): AhR expression in both CD4⁺ and CD8⁺ T cells is necessary for full suppression of a cytotoxic T lymphocyte response by TCDD

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Immunology and Microbiology

Neuroscience

Pharmacology, Toxicology and Pharmaceutical Science

T lymphocytes are direct, aryl hydrocarbon receptor (AhR)-dependent targets of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD): AhR expression in both CD4+ and CD8+ T cells is necessary for full suppression of a cytotoxic T lymphocyte response by TCDD

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Immunology and Microbiology

Neuroscience

Pharmacology, Toxicology and Pharmaceutical Science

T lymphocytes are direct, aryl hydrocarbon receptor (AhR)-dependent targets of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD): AhR expression in both CD4⁺ and CD8⁺ T cells is necessary for full suppression of a cytotoxic T lymphocyte response by TCDD